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1994 25
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The following term was not found in PubMed: 33 7979
Page 1
Genomic epidemiology of severe community-onset Acinetobacter baumannii infection.
Meumann EM, Anstey NM, Currie BJ, Piera KA, Kenyon JJ, Hall RM, Davis JS, Sarovich DS. Meumann EM, et al. Microb Genom. 2019 Mar;5(3):e000258. doi: 10.1099/mgen.0.000258. Epub 2019 Feb 26. Microb Genom. 2019. PMID: 30806611 Free PMC article.
We performed whole-genome sequencing on 55 clinical and five throat colonization A. baumannii isolates collected in northern Australia between 1994 and 2016. Clinical isolates included CAP (n=41), healthcare-associated pneumonia (n=7) and nosocomial bloodstream (n=7) isola …
We performed whole-genome sequencing on 55 clinical and five throat colonization A. baumannii isolates collected in northern Australia betwe …
Alzheimer's disease and transgenic mice.
Czech C, Masters C, Beyreuther K. Czech C, et al. J Neural Transm Suppl. 1994;44:219-30. doi: 10.1007/978-3-7091-9350-1_17. J Neural Transm Suppl. 1994. PMID: 7897394
Transgenic mice overexpressing the three major neuronal isoforms of the human amyloid precursor protein (APP), APP695, APP751, APP770 may provide an animal model for the analysis of the mechanisms and risk factors leading to amyloid deposition in Alzheimer's disease (AD) and D …
Transgenic mice overexpressing the three major neuronal isoforms of the human amyloid precursor protein (APP), APP695, APP751, APP770 may pr …
Transgenic mouse brain histopathology resembles early Alzheimer's disease.
Higgins LS, Holtzman DM, Rabin J, Mobley WC, Cordell B. Higgins LS, et al. Ann Neurol. 1994 May;35(5):598-607. doi: 10.1002/ana.410350514. Ann Neurol. 1994. PMID: 7513982
Here we show that the appearance and histological profile of deposits in the transgenic mice closely resemble those of preamyloid deposits in the brains of young adults with Down's syndrome, who presumably have the pathology of early-stage Alzheimer's disease. ...
Here we show that the appearance and histological profile of deposits in the transgenic mice closely resemble those of preamyloid deposits i …
Antenatal maternal serum profiles.
Spencer K. Spencer K. Br J Hosp Med. 1994 Mar 2-15;51(5):243-6. Br J Hosp Med. 1994. PMID: 7518729 Review.
More recently, biochemical screening programmes for chromosomal anomalies such as trisomy 21 (Down's syndrome) and trisomy 18 (Edward's syndrome) have moved on from the research stage and have begun to be implemented....
More recently, biochemical screening programmes for chromosomal anomalies such as trisomy 21 (Down's syndrome) and trisomy 18 (Edward …
Flow cytometric method to measure urea-resistant alkaline phosphatase from blood neutrophils for use in the prenatal screening of Down's syndrome.
Agrawal YP, Ylikangas P, Parviainen MT, Penttilä IM. Agrawal YP, et al. Prenat Diagn. 1994 Dec;14(12):1141-9. doi: 10.1002/pd.1970141207. Prenat Diagn. 1994. PMID: 7899281
The histochemical measurement of urea-resistant alkaline phosphatase from maternal blood neutrophils is known to have a high detection rate for the prenatal detection of Down's syndrome pregnancies. However, because the histochemical method is laborious and subjective to u …
The histochemical measurement of urea-resistant alkaline phosphatase from maternal blood neutrophils is known to have a high detection rate …
Fetal nuchal translucency: ultrasound screening for fetal trisomy in the first trimester of pregnancy.
Nicolaides KH, Brizot ML, Snijders RJ. Nicolaides KH, et al. Br J Obstet Gynaecol. 1994 Sep;101(9):782-6. doi: 10.1111/j.1471-0528.1994.tb11946.x. Br J Obstet Gynaecol. 1994. PMID: 7947527
It is predicted that for a false positive rate of 5%, the sensitivity of the new method of screening would be at least 85%, which compares favourably with the respective 20 to 30% and 50 to 60% of screening based on maternal age alone or the combination of maternal age with mater …
It is predicted that for a false positive rate of 5%, the sensitivity of the new method of screening would be at least 85%, which compares f …
beta-Amyloid protein immunoreactivity in skin is not a reliable marker of Alzheimer's disease. An autopsy-controlled study.
Heinonen O, Soininen H, Syrjänen S, Neittaanmäki H, Paljärvi L, Kosunen O, Syrjänen K, Riekkinen P Sr. Heinonen O, et al. Arch Neurol. 1994 Aug;51(8):799-804. doi: 10.1001/archneur.1994.00540200075019. Arch Neurol. 1994. PMID: 8042928
Aged nondemented controls were residents of a nursing home. The Down's syndrome (DS) group included both hospitalized and ambulatory patients. ...
Aged nondemented controls were residents of a nursing home. The Down's syndrome (DS) group included both hospitalized and ambulatory …
The primary structure of rat ribosomal protein L15.
Chang YL, Olvera J, Wool IG. Chang YL, et al. Biochem Biophys Res Commun. 1994 May 30;201(1):108-14. doi: 10.1006/bbrc.1994.1675. Biochem Biophys Res Commun. 1994. PMID: 8198562
Rat L15 has the hexapeptide, TYKFFE, that also occurs in the amyloidogenic glycoprotein A4 which is associated with Alzheimer's disease and Down's Syndrome....
Rat L15 has the hexapeptide, TYKFFE, that also occurs in the amyloidogenic glycoprotein A4 which is associated with Alzheimer's disease and …
Differential distribution of amyloid protein precursor immunoreactivity in the rat brain studied by using five different antibodies.
Beeson JG, Shelton ER, Chan HW, Gage FH. Beeson JG, et al. J Comp Neurol. 1994 Apr 1;342(1):78-96. doi: 10.1002/cne.903420109. J Comp Neurol. 1994. PMID: 8207129
The beta-amyloid or A4 protein is found deposited in neuritic plaques and neurofibrillary tangles in Alzheimer's disease (AD) affected brains and in the brains of adults with Down's Syndrome. The precursor to this 42 amino acid protein is the 695 amino acid long amyloid pr …
The beta-amyloid or A4 protein is found deposited in neuritic plaques and neurofibrillary tangles in Alzheimer's disease (AD) affected brain …
Microglia in diffuse plaques in hereditary cerebral hemorrhage with amyloidosis (Dutch). An immunohistochemical study.
Maat-Schieman ML, Rozemuller AJ, van Duinen SG, Haan J, Eikelenboom P, Roos RA. Maat-Schieman ML, et al. J Neuropathol Exp Neurol. 1994 Sep;53(5):483-91. doi: 10.1097/00005072-199409000-00007. J Neuropathol Exp Neurol. 1994. PMID: 7521904
Therefore, we studied the relationship between microglia and diffuse plaques in the cerebral cortex of four patients with HCHWA-D using a panel of macrophage/microglia markers (mAbs LCA, LeuM5, LeuM3, LN3, KP1, OKIa, CLB54, Mac1, Ki-M6, AMC30 and the lectin RCA-1). Eight AD patie …
Therefore, we studied the relationship between microglia and diffuse plaques in the cerebral cortex of four patients with HCHWA-D using a pa …
30 results